筛选
科学数据
统计数据
共检索到58条 ,权限内显示50条;
Data from: PPAR? promotes diabetes-associated centrosome amplification via increasing the expression of SKA1 directly at the transcriptional level
- 负责人：
- DOI：
- doi:10.5061/dryad.hg225q3
- 摘要：
- and insulin, and ROCK1 and 14-3-3? are signal mediators. In this study, we further investigated the molecular mechanisms of the centrosome amplification
Data from: Higher insulin resistance & adiposity in post-menopausal women with breast cancer treated with aromatase inhibitors
- 负责人：
- 关键词：
- DOI：
- doi:10.5061/dryad.2n054k4
- 摘要：
- Context: Aromatase deficiency causes obesity and insulin resistance in aromatase knockout mice and humans with rare mutations of the aromatase
Data from: Down-regulation of CXCL12/CXCR4 expression alleviates ischemia-reperfusion-induced inflammatory pain via inhibiting glial TLR4 activation
- 负责人：
- 关键词：
- DOI：
- doi:10.5061/dryad.5934k
- 摘要：
- Toll-like receptor 4 (TLR4) is important for the pathogenesis of inflammatory reactions and the promotion of pain processing after ischemia
Data from: Evolutionary conserved neural signature of early life stress affects animal social competence
- 负责人：
- Nyman, Cecilia
- 关键词：
- early environment mifepristone cooperative breeding stress axis glucocorticoid receptor cichlids
- DOI：
- doi:10.5061/dryad.47tc5
- 摘要：
- response 1) were associated with gr1 expression in the telencephalon and hypothalamus. When blocking glucocorticoid receptors (GR) with an antagonist, mifepristone
Human Lim Domain Kinase 1 (Limk1), Kinase Domain; A Target Enabling Package
- 负责人：
- 关键词：
- protein target enabling package disease structure cancer neuropsychiatry neuro neurological genetic disorders metabolic diseases oncology
- DOI：
- doi:10.5281/zenodo.1287260
- 摘要：
- via the kinase LIMK1. LIMK1 performs inhibitory phosphorylation on cofilin proteins blocking their actin-severing activity. Excessive BMPR2-LIMK1
Human Lim Domain Kinase 1 (Limk1), Kinase Domain; A Target Enabling Package
- 负责人：
- 关键词：
- protein target enabling package disease structure cancer neuropsychiatry neuro neurological genetic disorders metabolic diseases oncology
- DOI：
- doi:10.5281/zenodo.1241027
- 摘要：
- via the kinase LIMK1. LIMK1 performs inhibitory phosphorylation on cofilin proteins blocking their actin-severing activity. Excessive BMPR2-LIMK1
Human LIM domain kinase 1 (LIMK1), kinase domain; A Target Enabling Package
- 负责人：
- 关键词：
- protein target enabling package disease structure cancer neuropsychiatry neuro neurological genetic disorders metabolic diseases oncology
- DOI：
- doi:10.5281/zenodo.1219706
- 摘要：
- via the kinase LIMK1. LIMK1 performs inhibitory phosphorylation on cofilin proteins blocking their actin-severing activity. Excessive BMPR2-LIMK1
Data from: Regulatory mechanisms of group distributions in a gregarious arthropod
- 负责人：
- DOI：
- doi:10.5061/dryad.2v2m1
- 摘要：
- of local amplification processes, in agreement with the short-range activator and long-range inhibitor model (scale-dependent feedbacks). In other word
Supplementary Material for: NCOA3 Loss Disrupts Molecular Signature of Chondrocytes and Promotes Posttraumatic Osteoarthritis Progression
- 负责人：
- 关键词：
- Medicine
- DOI：
- doi:10.6084/m9.figshare.7140149
- 摘要：
- by siRNA or shRNA or inhibited by a chemical inhibitor to assess its role in chondrocyte dedifferentiation or OA pathogenesis in posttraumatic OA
Supplementary Material for: NCOA3 Loss Disrupts Molecular Signature of Chondrocytes and Promotes Posttraumatic Osteoarthritis Progression
- 负责人：
- 关键词：
- Medicine
- DOI：
- doi:10.6084/m9.figshare.7140149.v1
- 摘要：
- by siRNA or shRNA or inhibited by a chemical inhibitor to assess its role in chondrocyte dedifferentiation or OA pathogenesis in posttraumatic OA
