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Data from: A further cost for the sicker sex? Evidence for male-biased parasite-induced vulnerability to predation
负责人:
Jessica Stephenson
关键词:
parasite-induced vulnerability to predation sex-biased parasitism Bateman's principle for immunity infection tolerance
DOI:
doi:10.5061/dryad.3n6s7
摘要:
Males are typically the sicker sex. Data from multiple taxa indicate that they are more likely to be infected with parasites, and are less “tolerant
Data from: One stimulus - two responses: host and parasite life history variation in response to environmental stress
负责人:
关键词:
life-history evolution;Parasitism;Coevolution;Fecundity;Adaptation
DOI:
doi:10.5061/dryad.7bh53
摘要:
had significantly higher parasite reproductive outputs than infections in unstressed snails. The finding suggests that climate change may alt
Data from: Cross-species infection trials reveal cryptic parasite varieties and a putative polymorphism shared among host species.
负责人:
关键词:
Pasteuria ramosa;Daphnia longispina;Daphnia pulex;Parasitism;Coevolution;Daphnia magna
DOI:
doi:10.5061/dryad.vg928
摘要:
depends on the outcome of multiple steps and only some steps of the infection process may be critical in determining a parasites host range. To test
Data from: Genetic variation in resistance and fecundity tolerance in a natural host-pathogen interaction
负责人:
关键词:
Genetic Variation;Pandora neoaphidis;Parasitism;Fecundity;Acyrthosiphon pisum;Fitness
DOI:
doi:10.5061/dryad.24gq7
摘要:
t because there are multiple ways that organisms defend themselves against pathogens. One important distinction is between mechanisms of resistance tha
Data from: Protection against a fungal pathogen conferred by the aphid facultative endosymbionts Rickettsia and Spiroplasma is expressed in multiple
负责人:
关键词:
symbiosis;Host-parasite interaction;insects;Hamiltonella defensa;Spiroplasma;Rickettsia;Evolution of co-operation;Microbes;Sitobion avenae;Acyrthosiphon pisum
DOI:
doi:10.5061/dryad.81q1v
摘要:
ng host genotypes, but was not influenced by co-infection with Hamiltonella. Fecundity varied across single and double infections and between symbiont types, aphid
Data from: Co-infections and environmental conditions drive the distributions of blood parasites in wild birds
负责人:
Clark, Nicholas
关键词:
avian malaria immune modulation filarial parasite heterophil to lymphocyte ratio parasite co-occurrence
DOI:
doi:10.5061/dryad.pp6k4
摘要:
logistic regression. Co-infections occurred in 36% of infected birds. We identified both positively and negatively correlated parasite co-occurrence
Data from: Effector gene birth in plant parasitic nematodes: neofunctionalization of a housekeeping glutathione synthetase gene
负责人:
Eves-van den Akker, Sebastian
关键词:
Plant pathogen Effectors Evolution Genetic diversification Glutathione synthetase Thiol
DOI:
doi:10.5061/dryad.7vd0160
摘要:
) secrete proteins termed effectors into their host during infection to re-programme host development and immunity. The precise detail of how nematodes evolve
Phylogenetic analysis and virulence determinant of the host-adapted Staphylococcus aureus<\/i> lineage ST188 in China
负责人:
关键词:
Microbiology 59999 Environmental Sciences not elsewhere classified Ecology 69999 Biological Sciences not elsewhere classified Cancer 110309 Infectious Diseases Plant Biology 60506 Virology
DOI:
doi:10.6084/m9.figshare.7937831
摘要:
-sensitive S. aureus (MSSA) ST188 as the major lineage causing infections in multiple host species. Whole-genome sequencing and phenotypic analyses
Data from: Diversity and distribution of Wolbachia in relation to geography, host plant affiliation and life cycle of a heterogonic gall wasp
负责人:
Schuler, Hannes
关键词:
endosymbiont speciation wsp
DOI:
doi:10.5061/dryad.6k94r
摘要:
-infection in Georgia and triple-infections in two populations in central Florida. Conclusions: The diversity of Wolbachia across geographically
Data from: Risk alleles for tuberculosis infection associate with reduced immune reactivity in a wild mammalian host
负责人:
Tavalire, Hannah
关键词:
Ecological genetics ecoimmunology
DOI:
doi:10.5061/dryad.3c45k28
摘要:
production indicative of variation in macrophage activation. Here we provide measurable differences in infection resistance at multiple

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