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Data from: Local adaptation at higher trophic levels: contrasting hyperparasite-pathogen infection dynamics in the field and laboratory
负责人:
关键词:
Hyperparasite;disease;Host Parasite Interactions;local adaptation;Coevolution;Podosphaera plantaginis
DOI:
doi:10.5061/dryad.5fh23
摘要:
of local adaptation in disease systems have mostly focused on interactions between competing pathogens or pathogens and their hosts. In nature, parasites and pathogens
Data from: Genetic variation in resistance and fecundity tolerance in a natural host-pathogen interaction
负责人:
关键词:
Genetic Variation;Pandora neoaphidis;Parasitism;Fecundity;Acyrthosiphon pisum;Fitness
DOI:
doi:10.5061/dryad.24gq7
摘要:
fungal pathogens. However, it is unclear whether aphid genotypes vary in defense against Pandora in the absence of protective symbionts. We therefore measured
Data from: Immune priming specificity within and across generations reveals the range of pathogens affecting evolution of immunity in an insect
负责人:
Dhinaut, Julien
关键词:
host-pathogen interaction ecological immunology invertebrates maternal effects immune priming transgenerational immune priming
DOI:
doi:10.5061/dryad.t2850
摘要:
s costly. We concluded that Gram-positive bacterial pathogens were of great importance in the evolution of immune priming in this insect species.
Data from: Genotype specificity among hosts, pathogens, and beneficial microbes influences the strength of symbiont mediated protection
负责人:
关键词:
Fungal pathogens;Regiella insecticola;endosymbiont;Pandora neoaphidis;pea aphid;Coevolution;Symbiont-mediated resistance;mutualism;Acyrthosiphon pisum
DOI:
doi:10.5061/dryad.6q750
摘要:
d symbiont of an aphid is influenced by both host-symbiont and symbiont-pathogen genotype by genotype interactions. Further, we show that certain symbiont
Data from: The mammalian complement system as an epitome of hostpathogen genetic conflicts
负责人:
关键词:
human specific infections;molecular evolution;host-pathogen genetic conflict;Complement system;positive selection
DOI:
doi:10.5061/dryad.5jt25
摘要:
-interacting proteins. Complement components targeted by several pathogens evolved under strong selective pressure in primates, with selection acting on residues
Data from: Pre-infection effects of nectar secondary compounds on a bumble bee gut pathogen
负责人:
关键词:
pathogens;Crithidia bombi;Insect-Parasitoid Interactions;Bombus impatiens;floral chemistry;Apidae;insect-plant interactions
DOI:
doi:10.5061/dryad.4gg142s
摘要:
y be deposited in floral nectar, providing a site for pathogens to interact with nectar secondary compounds prior to infecting bees. Some nect
Data from: Linking sex differences to the evolution of infectious disease life-histories
负责人:
Hall, Matthew D.
关键词:
sexual dimorphism host–pathogen interactions host heterogeneity pathogen evolution sexual selection
DOI:
doi:10.5061/dryad.s7n1b44
摘要:
the lower performance of a pathogen within a male host, making even subtle differences between the sexes evolutionarily relevant, as long as the selection generate
Data from: Fatal diseases and parasitoids: from competition to facilitation in a shared host.
负责人:
关键词:
emerging infectious disease;Co-infection;nucleopolyhedrovirus;parasite interactions;Entomophaga maimaiga;population dynamics;Lymantria dispar;Cotesia melanoscela
DOI:
doi:10.5061/dryad.3tn08
摘要:
ve exclusion to facilitation, can drive community structure and dynamics. Emergent pathogens have the potential to greatly alter community interactions. We found tha
Data from: Rapid evolution of microbe-mediated protection against pathogens in a worm host
负责人:
关键词:
DOI:
doi:10.5061/dryad.nd848
摘要:
king. Using experimental evolution of a novel, tripartite interaction, we demonstrate that mildly pathogenic bacteria (Enterococcus faecalis) living in worms
Data from: Evolution of both host resistance and tolerance to an emerging bacterial pathogen
负责人:
关键词:
host-parasite interactions;Coevolution;Haemorrhous mexicanus;Adaptation
DOI:
doi:10.5061/dryad.4711h78
摘要:
e so accounting for significant natural variation in pathogen virulence, despite known effects on host responses to infection. Here, we investigate whether

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