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Data from: The mammalian complement system as an epitome of host–pathogen genetic conflicts
- 负责人：
- DOI：
- doi:10.5061/dryad.5jt25
- 摘要：
- lico mutagenesis and protein–protein docking analyses supported this by showing that positively selected sites, both in the host's and in the pathogen's interacting
Data from: Rapid antagonistic coevolution in an emerging pathogen and its vertebrate host
- 负责人：
- DOI：
- doi:10.5061/dryad.5g5n8n2
- 摘要：
- , with hosts and pathogens displaying increased resistance and virulence in response to each other over time.
Data from: Soft selective sweeps in fungicide resistance evolution: recurrent mutations without fitness costs in grapevine downy mildew
- 负责人：
- DOI：
- doi:10.5061/dryad.vp8n9
- 摘要：
- fungicide resistance has evolved in this plant pathogen, to determine whether hard or soft selective sweeps were involved. An analysis of nucleotide polymorphism in 108
Data from: Nonparallelism in MHC II? diversity accompanies nonparallelism in pathogen infection of lake whitefish (Coregonus clupeaformis) species
- 负责人：
- DOI：
- doi:10.5061/dryad.ft94b
- 摘要：
- Major histocompatibility (MHC) immune system genes may evolve in response to pathogens in the environment. Because they also may affect mate choice
Data from: Inferring epidemiologic dynamics from viral evolution: 2014–2015 Eurasian/North American highly pathogenic avian influenza viruses exceed
- 负责人：
- DOI：
- doi:10.5061/dryad.247st
- 摘要：
- into wild and domestic bird hosts: (i) transmission of novel HPAIVs in wild birds was restricted by mechanisms associated with highly-pathogenic
Data from: A metatranscriptomic analysis of diseased social wasps (Vespula vulgaris) for pathogens, with an experimental infection of larvae
- 负责人：
- DOI：
- doi:10.5061/dryad.m98pt3k
- 摘要：
- disease. First, next-generation sequencing and metatranscriptomic analysis were used to examine pathogen presence. The transcriptome of healthy and diseased
Data from: Host ecotype generates evolutionary and epidemiological divergence across a pathogen metapopulation
- 负责人：
- 关键词：
- DOI：
- doi:10.5061/dryad.vf33q
- 摘要：
- The extent and speed at which pathogens adapt to host resistance varies considerably. This presents a challenge for predicting when—and wher
Data from: Immunogenetic novelty confers a selective advantage in host-pathogen coevolution
- 负责人：
- DOI：
- doi:10.5061/dryad.72262
- 摘要：
- known. Its high diversity is usually attributed to selection imposed by fast-evolving pathogens. Pathogens are thought to evolve to escape recognition by common
Data from: CAG repeat not polyglutamine length determines timing of Huntington’s disease onset
- 负责人：
- DOI：
- doi:10.5061/dryad.5d4s2r8
- 摘要：
- naturally occurring genetic modifier loci, defined by GWAS, may influence HD pathogenesis through other mechanisms. These findings have profound implications
Data from: Epistasis buffers the fitness effects of rifampicin-resistance mutations in Pseudomonas aeruginosa
- 负责人：
- DOI：
- doi:10.5061/dryad.8858
- 摘要：
- tance in pathogen populations by determining the fitness cost associated with resistance. We used an experimental evolution approach to tes
