RAS-modification as a novel therapy for pulmonary indications in T2D

负责人：

关键词：

Clinical and Experimental Therapeutics T2DM angiotensin(1-7) immune function lung fibrosis pulmonary indications

DOI：

doi:10.25549/usctheses-c40-350071

摘要：

of renin-angiotensin system (RAS) results in increased levels of A-II and signaling through AT1 receptor. This pathway is well recognized for its role in induction of OS