Data from: Influence of hepatitis C virus and IL28B genotypes on liver stiffness

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关键词：

hepatitis C genotypes;Fibrosis progression;single nucleotide polymorphism (SNP);transient elastography;IL28b genotypes;liver cirrhosis

DOI：

doi:10.5061/dryad.0q524

摘要：

s for fibrosis and cirrhosis, only increasing age (odds ratio (OR) 1.09 (95% confidence interval (CI), 1.05–1.14 per year increment)), ALT (OR 1.01 (95% CI, 1.002

Data from: Early predictors of outcomes of hospitalization for cirrhosis and assessment of the impact of race and ethnicity at safety-net hospitals

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关键词：

Cirrhosis;hospitalization;readmission;mortality;2012;Safety-net hospital;Cirrhosis

DOI：

doi:10.5061/dryad.6gt88dv

摘要：

with cirrhosis are relatively understudied and may be vulnerable to poor outcomes and racial/ethnic disparities. Aims. To examine the outcomes of patients

Data from: Long-term benefit of hepatitis C therapy in a safety net hospital system: a cross-sectional study with median 5-year follow-up

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关键词：

Public health;Infectious Diseases;Health economics;Hepatology;Hepatobiliary disease

DOI：

doi:10.5061/dryad.qc57j

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(OR 0.20, 95% CI 0.07 – 0.54), genotype 1 HCV infection (OR 0.25, 95% CI 0.13 – 0.50) and the presence of cirrhosis (OR 0.26, 95% CI 0.12 – 0.58). Survival

Data from: Systematic review: unmet supportive care needs in people diagnosed with chronic liver disease

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关键词：

Hepatology;Hepatobiliary disease;Public health;Epidemiology

DOI：

doi:10.5061/dryad.5jn76

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Objective: People with chronic liver disease, particularly those with decompensated cirrhosis, experience several potentially debilitating

for hepatocellular carcinoma

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关键词：

osteopontin;alpha-fetoprotein;dickkopf-1;Hepatocellular carcinoma;PIVKA-II

DOI：

doi:10.5061/dryad.3n901

摘要：

and 193 liver cirrhosis control patients, plasma AFP, PIVKA-II, OPN and DKK-1 levels were measured by ELISA, and r

Data from: Widespread dysregulation of long non-coding genes associated with fatty acid metabolism, cell division, and immune response gene

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DOI：

doi:10.5061/dryad.xksn02vbn

摘要：

d metabolism, cell division, or immune response pathways, and with apoptosis or cirrhosis. We also identified hub and bottleneck lncRNAs, which are expected to be key regulator

Hepatitis B virus X protein regulation of β-catenin and NANOG and co-regulatory role with YAP1 in HCC malignancy

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关键词：

Molecular Microbiology and Immunology hepatocellular carcinoma HCC hepatitis B virus X protein HBx NANOG YAP1 β-catenin tumor initiating cells TICs

DOI：

doi:10.25549/usctheses-c3-615884

摘要：

liver cirrhosis and constant inflammation. As sorafenib treatment is not curative, tumor-initiating stem-like cells (TICs) contributes to the low survival rates